Connect with us

Science & Technology

Prominent Yale Professor Explains How Darwin’s Theory of Evolution Doesn’t Match The Science

  • The Facts:Darwin’s Theory of Evolution has, for a great many scientists, become relatively obsolete in the face of new research into the creation and generation of life.
  • Reflect On:Can we see that the belief in the randomness of the creation and evolution of life, as posited by Darwin’s Theory of Evolution, is a limitation on human progress and no longer serving us in our collective evolution?

Science never ceases to question. When a theory is taught as an unquestionable fact, it should be quite obvious that something is wrong. Today, science isn’t really science, and this is not only true for topics such as evolution, it’s true in many areas where science is used for an agenda by powerful and corrupt forces.

Health sciences are a great example. As Bud Relman, former editor of the New England Journal of Medicine said, “The medical profession is being bought by the pharmaceutical industry, not only in terms of the practice of medicine, but also in terms of teaching and research. The academic institutions of this country are allowing themselves to be the paid agents of the pharmaceutical industry. I think it’s disgraceful.”

Today, some scientific publications are silenced and others are pushed forward, depending on how they affect corporate and political agendas. It’s not actually about the science. What the mainstream media preaches as “settled science” is not actually settled. In fact it is often highly dubious. Why don’t more people see this? The answer is simple, it’s because we rely on outside sources to tell us ‘what is,’ instead of taking the time, as individual researchers, to really look into something.

The Theory Of Evolution

The ‘Theory of Evolution’ falls into this category. Scientists who have rejected the basic premises of Darwin’s theory continue to be condemned and shunned by the mainstream community and powerful people. This is because their paradigm-shifting thoughts and ideas on the subject, though more grounded in fact, threaten the goal of the global elite, which NSA whistleblower William Binney says, is “total population control.” The average person who gets a bachelor’s degree in science is trained to simply repeat the same old textbook rhetoric as to why evolution is the be all and end all of human existence, without actually looking into why the theory is highly questionable.

One of the latest dissenters is David Gelernter, a prominent scientist and distinguished professor of computer science at Yale University. He recently published an essay in the Claremont Review of Books explaining his objections to a premise behind Darwin’s theory.

He first points to the famous “Cambrian Explosion” which occurred half a billion years ago, in which a number of new organisms, including the first ever known animals, pop up suddenly in the fossil record over a period of approximately 70 million years. Apparently, this giant explosion of spontaneous life was followed by evolution, slow growth and “scanty fossils, mainly of single celled organisms, dating back to the origins of life roughly three and a half billions years ago.”

From here, he explains how Darwin’s theory predicts that new life forms evolve gradually from preceding ones. but if this is applied to the Cambrian creatures as well, it doesn’t work. The predecessors to the Cambrian creatures are missing, something that Darwin himself was disturbed by as well. Furthermore, even without this fact, many scientists have already used other aspects of the fossil record to demonstrate that Darwin’s theory is clearly wrong.

The Cambrian explosion had been unearthed, and beneath those Cambrian creatures their Precambrian predecessors should have been waiting – and weren’t. In fact, the fossil record as a whole lacked the upward-branching structure Darwin predicted….the ever-expanding fossil archives don’t look good for Darwin, who made clear and concrete predictions that have (so far) been falsified—according to many reputable paleontologists, anyway. When does the clock run out on those predictions? Never. But any thoughtful person must ask himself whether scientists today are looking for evidence that bears on Darwin, or looking to explain away evidence that contradicts him. There are some of each. Scientists are only human, and their thinking (like everyone else’s) is colored by emotion. (source)

The Genesis Of New Life Forms

His next point goes a little deeper. Many people point to the fact that variation occurs naturally among individuals and different traits are past on, this is something observable and something that we all know. Many scientists actually use this point as a proof for evolution, which doesn’t make much sense. According to proponents of the theory of evolution, natural variation is the consequence of random change or mutation to cells, to the genetic information within our cells that deal with reproduction. These cells pass on genetic change to the next generation, which, according to Darwinians, changes the future of the species and not just the individual.

The engine behind this thought, as Gelernter explains, is ‘change’ driven by the survival of the fittest and, obviously, lots and lots of time. He then goes on to ask a very crucial question: What exactly does generating new forms of life entail? Many within the field agree that generating a new shape of protein is the key to it. But does Darwinian evolution even purport to be able to do that? For Chris Williams, A Ph.D., Biochemistry Ohio State University, the full scope of Darwinian Evolution barely touches upon this important matter:

As a biochemist and software developer who works in genetic and metabolic screening, I am continually amazed by the incredible complexity of life. For example, each of us has a vast ‘computer program’ of six billion DNA bases in every cell that guided our development from a fertilized egg, specifies how to make more than 200 tissue types, and ties all this together in numerous highly functional organ systems. Few people outside of genetics or biochemistry realize that evolutionists still can provide no substantive details at all about the origin of life, and particularly the origin of genetic information in the first self-replicating organism. What genes did it require — or did it even have genes? How much DNA and RNA did it have — or did it even have nucleic acids? How did huge information-rich molecules arise before natural selection? Exactly how did the genetic code linking nucleic acids to amino acid sequence originate? Clearly the origin of life — the foundation of evolution – is still virtually all speculation, and little if no fact.

Intelligent Design

More and more, the evidence points to the great intelligence apparent in the system of life-creation. The reason that Darwinian Evolution is being left behind, and for many is obsolete, is because it is completely based on random, non-intelligent processes. Edward Peltzer Ph.D. Oceanography, University of California, San Diego (Scripps Institute), Associate Editor, Marine Chemistry, uses a clear real-life laboratory example to explain the need to posit the existence of an overriding ‘intelligence’ in order for things to make any sense:

As a chemist, the most fascinating issue for me revolves around the origin of life. Before life began, there was no biology, only chemistry — and chemistry is the same for all time. What works (or not) today, worked (or not) back in the beginning. So, our ideas about what happened on Earth prior to the emergence of life are eminently testable in the lab. And what we have seen thus far when the reactions are left unguided as they would be in the natural world is not much. Indeed, the decomposition reactions and competing reactions out distance the synthetic reactions by far. It is only when an intelligent agent (such as a scientist or graduate student) intervenes and “tweaks” the reactions conditions “just right” do we see any progress at all, and even then it is still quite limited and very far from where we need to get. Thus, it is the very chemistry that speaks of a need for something more than just time and chance. And whether that be simply a highly specified set of initial conditions (fine-tuning) or some form of continual guidance until life ultimately emerges is still unknown. But what we do know is the random chemical reactions are both woefully insufficient and are often working against the pathways needed to succeed. For these reasons I have serious doubts about whether the current Darwinian paradigm will ever make additional progress in this area.

Gelernter brings this conversation specifically to the generation of proteins:

Proteins are the special ops forces (or maybe the Marines) of living cells, except that they are common instead of rare; they do all the heavy lifting, all the tricky and critical assignments, in a dazzling range of roles. Proteins called enzymes catalyze all sorts of reactions and drive cellular metabolism. Other proteins (such as collagen) give cells shape and structure, like tent poles but in far more shapes. Nerve function, muscle function, and photosynthesis are all driven by proteins. And in doing these jobs and many others, the actual, 3-D shape of the protein molecule is important.

So, is the simple neo-Darwinian mechanism up to this task? Are random mutation plus natural selection sufficient to create new protein shapes?

Diving Into Proteins

Gelernter goes on to answer that question in great detail, and after going through the entire explanation he comes to what seems to be an inarguable conclusion. That the Theory of Evolution cannot, in any way, be a possible explanation for the generation of new proteins and mutations that are required for evolution to occur at all. This explanation is complex, but well worth it if you really want to understand how the ‘Theory of Evolution’ is refuted by the science of proteins:

How to make proteins is our first question. Proteins are chains: linear sequences of atom-groups, each bonded to the next. A protein molecule is based on a chain of amino acids; 150 elements is a “modest-sized” chain; the average is 250. Each link is chosen, ordinarily, from one of 20 amino acids. A chain of amino acids is a polypeptide—“peptide” being the type of chemical bond that joins one amino acid to the next. But this chain is only the starting point: chemical forces among the links make parts of the chain twist themselves into helices; others straighten out, and then, sometimes, jackknife repeatedly, like a carpenter’s rule, into flat sheets. Then the whole assemblage folds itself up like a complex sheet of origami paper. And the actual 3-D shape of the resulting molecule is (as I have said) important.

Imagine a 150-element protein as a chain of 150 beads, each bead chosen from 20 varieties. But: only certain chains will work. Only certain bead combinations will form themselves into stable, useful, well-shaped proteins.

So how hard is it to build a useful, well-shaped protein? Can you throw a bunch of amino acids together and assume that you will get something good? Or must you choose each element of the chain with painstaking care? It happens to be very hard to choose the right beads.

Inventing a new protein means inventing a new gene. (Enter, finally, genes, DNA etc., with suitable fanfare.) Genes spell out the links of a protein chain, amino acid by amino acid. Each gene is a segment of DNA, the world’s most admired macromolecule. DNA, of course, is the famous double helix or spiral staircase, where each step is a pair of nucleotides. As you read the nucleotides along one edge of the staircase (sitting on one step and bumping your way downwards to the next and the next), each group of three nucleotides along the way specifies an amino acid. Each three-nucleotide group is a codon, and the correspondence between codons and amino acids is the genetic code. (The four nucleotides in DNA are abbreviated T, A, C and G, and you can look up the code in a high school textbook: TTA and TTC stand for phenylalanine, TCT for serine, and so on.)

Your task is to invent a new gene by mutation—by the accidental change of one codon to a different codon. You have two possible starting points for this attempt. You could mutate an existing gene, or mutate gibberish. You have a choice because DNA actually consists of valid genes separated by long sequences of nonsense. Most biologists think that the nonsense sequences are the main source of new genes. If you tinker with a valid gene, you will almost certainly make it worse—to the point where its protein misfires and endangers (or kills) its organism—long before you start making it better. The gibberish sequences, on the other hand, sit on the sidelines without making proteins, and you can mutate them, so far as we know, without endangering anything. The mutated sequence can then be passed on to the next generation, where it can be mutated again. Thus mutations can accumulate on the sidelines without affecting the organism. But if you mutate your way to an actual, valid new gene, your new gene can create a new protein and thereby, potentially, play a role in evolution.

Mutations themselves enter the picture when DNA splits in half down the center of the staircase, thereby allowing the enclosing cell to split in half, and the encompassing organism to grow. Each half-staircase summons a matching set of nucleotides from the surrounding chemical soup; two complete new DNA molecules emerge. A mistake in this elegant replication process—the wrong nucleotide answering the call, a nucleotide typo—yields a mutation, either to a valid blueprint or a stretch of gibberish.

Building a Better Protein

Now at last we are ready to take Darwin out for a test drive. Starting with 150 links of gibberish, what are the chances that we can mutate our way to a useful new shape of protein? We can ask basically the same question in a more manageable way: what are the chances that a random 150-link sequence will create such a protein? Nonsense sequences are essentially random. Mutations are random. Make random changes to a random sequence and you get another random sequence. So, close your eyes, make 150 random choices from your 20 bead boxes and string up your beads in the order in which you chose them. What are the odds that you will come up with a useful new protein?

It’s easy to see that the total number of possible sequences is immense. It’s easy to believe (although non-chemists must take their colleagues’ word for it) that the subset of useful sequences—sequences that create real, usable proteins—is, in comparison, tiny. But we must know how immense and how tiny.

The total count of possible 150-link chains, where each link is chosen separately from 20 amino acids, is 20150. In other words, many. 20150 roughly equals 10195, and there are only 1080 atoms in the universe.

What proportion of these many polypeptides are useful proteins? Douglas Axe did a series of experiments to estimate how many 150-long chains are capable of stable folds—of reaching the final step in the protein-creation process (the folding) and of holding their shapes long enough to be useful. (Axe is a distinguished biologist with five-star breeding: he was a graduate student at Caltech, then joined the Centre for Protein Engineering at Cambridge. The biologists whose work Meyer discusses are mainly first-rate Establishment scientists.) He estimated that, of all 150-link amino acid sequences, 1 in 1074 will be capable of folding into a stable protein. To say that your chances are 1 in 1074 is no different, in practice, from saying that they are zero. It’s not surprising that your chances of hitting a stable protein that performs some useful function, and might therefore play a part in evolution, are even smaller. Axe puts them at 1 in 1077.

In other words: immense is so big, and tiny is so small, that neo-Darwinian evolution is—so far—a dead loss. Try to mutate your way from 150 links of gibberish to a working, useful protein and you are guaranteed to fail. Try it with ten mutations, a thousand, a million—you fail. The odds bury you. It can’t be done.

Proteins/Mutations Are One of Several Issues

Despite all of the scientific dogma that plagues this issue, proteins/mutations and lack of fossil evidence are simply the tip of the iceberg when it comes to finding faults found within the Theory of Evolution. There are many facts, information, science and new discoveries that would make one wonder how it’s even still being taught.

Furthermore, despite the fact that we get pounded with the idea that random mutation is ultimate truth within the mainstream, and that one is wrong for questioning it, there are a number of prominent scientists, who are actually getting together in large numbers to collectively refute Darwinism. A group of 500 scientists from several fields came together a few years to create “A Scientific Dissent From Darwinism,” as one examples. The issue is that these scientists are never getting any mainstream attention. But clearly there are some very intelligent people here.

The theory will be with us for a long time, exerting enormous cultural force. Darwin is no Newton. Newton’s physics survived Einstein and will always survive, because it explains the cases that dominate all of space-time except for the extreme ends of the spectrum, at the very smallest and largest scales. It’s just these most important cases, the ones we see all around us, that Darwin cannot explain. Yet his theory does explain cases of real significance. And Darwin’s intellectual daring will always be inspiring. The man will always be admired.

He now poses a final challenge. Whether biology will rise to this last one as well as it did to the first, when his theory upset every apple cart, remains to be seen. How cleanly and quickly can the field get over Darwin, and move on?—with due allowance for every Darwinist’s having to study all the evidence for himself? There is one of most important questions facing science in the 21st century.

Other Examples That Throw Off The Theory Of Evolution

Not long ago I wrote about a  recent paper published by 33 scientists in the Progress in Biophysics and Molecular Biology journal suggesting that the flourishing of life during the Cambrian era (Cambrian Explosion) originates from the stars is so fascinating.

“With the rapidly increasing number of exoplanets that have been discovered in the habitable zones of long-lived red dwarf stars (Gillon et al., 2016), the prospects for genetic exchanges between life-bearing Earth-like planets cannot be ignored. ” (The study)

There is a great little blurb from Cosmos Magazine, one of the few outlets who are talking about the study:

With 33 authors from a wide range of reputable universities and research institutes, the paper makes a seemingly incredible claim. A claim that if true, would have the most profound consequences for our understanding of the universe. Life, the paper argues, did not originate on the planet Earth.

The response?

Near silence.

The reasons for this are as fascinating as the evidence and claims advanced by the paper itself. Entitled “Cause of the Cambrian Explosion – Terrestrial or Cosmic?”, the publication revives a controversial idea concerning the origin of life, an idea stretching back to Ancient Greece, known as ‘panspermia.a’.

Academics like Francis Crick, an English scientist who co-discovered the structure of the DNA molecule (alongside James D. Watson), argues that there is no possible way that the DNA molecule could have originated on Earth. The generally accepted theory in this field, as explained above, is that we are the result of a bunch of molecules accidentally bumping into each other, creating life. However, according to Crick, we are the result of what is now known as Directed Panspermia. Crick and British chemist Leslie Orgel published their paper on it in July of 1973, hinting that we were brought here by chance, or by some sort of intelligence from somewhere else in the universe.

This is interesting, because then you can get into the lore of creation stories that exists within ancient cultures from around the world, one would be our relation to, for example, what many indigenous culture refer to as the ‘Star People.’

I’m not even going to go into all of the strange skeletal remains that have been completely left out of the record, like the remains of giants, for example.

The Takeaway

The agenda for the maintenance of the neo-Darwinian version of the ‘Theory of Evolution’ was nothing less than to move people away from the notion of an intelligent creator and towards a perception founded in scientific materialism. In this way, those who funded and controlled scientific activity on the planet would have tremendous power.

Darwin’s theory may have served humanity for a certain phase of our own evolution, but now it is holding us back. It’s time for all of us to pierce more deeply into an understanding of the nature of the creation of life if we are to become creators ourselves by studying the current evidence. As the group of 500 scientists asked, ‘How cleanly and quickly can the field get over Darwin, and move on?—with due allowance for every Darwinist’s having to study all the evidence for himself?’

Source link

Advertisement
Comments

Science & Technology

This is the world’s first commercial flying car

The world’s first commercial flying car is already on sale. It is equipped with two retractable propellers and rear wings.

The vehicle was presented during the Miami Art Week 2019 by the Dutch company PAL-V International. It is called Liberty, and its price is around 600,000 dollars.

It has Dutch engineering and Italian design, it is already in active production and has at least 70 anticipated.

“As soon as Nicolas Cugnot invented the car and the Wright brothers made their first successful flight, people began to dream of combining the two in a flying car.”

‘It turned out to be more complicated than initially estimated: a complex puzzle. However, once resolved, it would create maximum freedom in mobility’, said the executive director of the company, Robert Dingemanse.

PAL-V Flying car "width =" 780 "height =" 390 "
Credit: pal-v.com

When will it be available?

The first units are expected to reach their owners in 2021. However, it must be borne in mind that to handle it, it is necessary to have not only the driver’s license, but also the pilot’s license.

The new car has two versions, the Pioneer and the sports version. Robert Dingemanse explained that the Pioneer version differs from Liberty by its a complete carbon package. He also revealed that only 90 flying cars will be manufactured in this version.

Features of the flying car

Flying car "width =" 1100 "height =" 619 "srcset ="
PAL-V Pioneer. Credit: pal-v.com
Inside of the flying car "width =" 1104 "height =" 736 "srcset ="
Interior of the flying car. Credit: pal-v.com

The PAL-V, a three-wheeled vehicle that can carry up to two passengers and 20 kilos of cargo, is basically a hybrid between a car and a helicopter.

According to the company website, the PAL-V has a four-cylinder engine and is capable of flying at an altitude of up to 3,500 meters. The vehicle, which is made with carbon fiber, titanium and aluminum and weighs only 664 kilograms, uses gasoline for cars and can reach maximum speeds of 180 km / h in the air and 160 km / h on land.

It also has both a ground and air system similar to that of a motorcycle in which the pilot-driver tilts the machine with a control lever.

It also stands out that the PAL-V converts from car to gyrocopter in just 10 minutes and can accelerate from 0 to 100 km / h in less than 9 seconds.

Continue Reading

Science & Technology

Health authorities have confirmed a case of a rare type of smallpox in a UK patient

Skin rashes caused by ape pox. Credit: CDC's Public Health Image Library (Public domain)

A patient in England has been diagnosed with a rare case of monkeypox, as reported by Public Health England (PHE).

The rare viral infection is similar to smallpox, and though it is milder, it can be fatal.

It has been reported that the individual was in Nigeria and that he would have contracted the disease there. Later, upon returning to the United Kingdom, he stayed in the southwest of England where the disease occurred.

Upon symptoms, he was transferred to the Guy’s and St Thomas’ NHS Foundation Trust , a center specializing in infectious diseases in London.

The health authorities have taken the necessary measures to prevent the virus from spreading to other people.

Vaccination against smallpox to people in Africa. (Public domain)

The PHE said in a statement:

As a precaution, PHE experts are working closely with NHS colleagues to implement rapid infection control procedures, including contact with people who may have been in close contact with the individual to provide health information and advice. ”

But experts are not very worried about contagion, because monkeypox does not spread easily among people and the risk of affecting the population is quite low, said Dr. Meera Chand , PHE consulting microbiologist.

This transmission electron micrograph (TEM) represents a series of smallpox virus virions. Credit: CDC / Dr. Fred Murphy; Sylvia Whitfield / Wikimedia Commons

Although the infection usually occurs mildly and people get better without treatment; Some individuals may develop very serious symptoms, with a percentage of 1 to 10 percent of patients dying from the disease during outbreaks, according to the World Health Organization .

The symptoms presented are similar to those of smallpox but milder. First, fever, headaches, muscle aches, back pain, swollen lymph nodes, chills and exhaustion. Subsequently rashes may appear on the skin , starting on the face and spreading throughout the rest of the body.

This is not the first time a patient has been infected with smallpox in the United Kingdom. In 2018, there were three cases after a person was diagnosed with the disease. The individual had also returned from Nigeria.

Source: Gov.ukIFL Science

Continue Reading

Science & Technology

A cold virus can infect a pregnant woman’s fetus

The study showed that the expectant mother is able to transmit a respiratory tract infection to her unborn child.

Scientists from Tulane University (Louisiana, USA) received the first evidence that the cold virus, which affects a pregnant woman, can penetrate the placenta and infect the fetus. An article about this has been published in PLOS One .

The placenta, an organ that develops in the uterine cavity of a woman during pregnancy, provides the necessary nutrition from the mother to the embryo and simultaneously performs another important task: it filters out potential pathogenic microorganisms. However, a group of pediatricians led by Professor Giovanni Piedimonte found that this natural “barrier” is not so impenetrable.

Scientists took the placenta from donors, isolated three main types of cells – cytotrophoblasts, fibroblasts and Kashchenko – Hofbauer cells – and in vitro exposed them to the human respiratory syncytial virus, which causes respiratory tract infections. Although cytotrophoblast cells supported a weak process of the spread of the virus, two other types were more susceptible to infection. So, Kashchenko-Hofbauer cells survived and allowed the virus to replicate inside the cell walls. According to scientists, then these cells, moving inside the placenta, are able to transmit the virus to the fetus.

“Such cells do not die after they become infected,” Piedimonte explains. – When they enter the fetus, they are comparable to bombs stuffed with a virus. They do not spread the virus in the area of ​​the “explosion”, but carry it through the intercellular channels. <…> Thus, our theory is confirmed that when a woman gets a cold during pregnancy, the virus that causes the infection can pass to the fetus and cause a pulmonary infection before the birth of a child. ”

Pediatricians also suggested that the respiratory syncytial virus is able to infect the lung tissue of the unborn baby and provoke the development of an infection that will subsequently affect the predisposition to asthma. To confirm or refute their theory, the authors of the study intend to conduct clinical tests.

Last year, scientists from the University of Cambridge created an artificial and functional mini-placenta using trophoblasts, and recently it turned out that particles of air pollution can penetrate the placenta of pregnant women

Continue Reading

Recent Comments

Trending