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Prominent Yale Professor Explains How Darwin’s Theory of Evolution Doesn’t Match The Science

Prominent Yale Professor Explains How Darwin’s Theory of Evolution Doesn’t Match The Science 86
  • The Facts:Darwin’s Theory of Evolution has, for a great many scientists, become relatively obsolete in the face of new research into the creation and generation of life.
  • Reflect On:Can we see that the belief in the randomness of the creation and evolution of life, as posited by Darwin’s Theory of Evolution, is a limitation on human progress and no longer serving us in our collective evolution?

Science never ceases to question. When a theory is taught as an unquestionable fact, it should be quite obvious that something is wrong. Today, science isn’t really science, and this is not only true for topics such as evolution, it’s true in many areas where science is used for an agenda by powerful and corrupt forces.

Health sciences are a great example. As Bud Relman, former editor of the New England Journal of Medicine said, “The medical profession is being bought by the pharmaceutical industry, not only in terms of the practice of medicine, but also in terms of teaching and research. The academic institutions of this country are allowing themselves to be the paid agents of the pharmaceutical industry. I think it’s disgraceful.”

Today, some scientific publications are silenced and others are pushed forward, depending on how they affect corporate and political agendas. It’s not actually about the science. What the mainstream media preaches as “settled science” is not actually settled. In fact it is often highly dubious. Why don’t more people see this? The answer is simple, it’s because we rely on outside sources to tell us ‘what is,’ instead of taking the time, as individual researchers, to really look into something.

The Theory Of Evolution

The ‘Theory of Evolution’ falls into this category. Scientists who have rejected the basic premises of Darwin’s theory continue to be condemned and shunned by the mainstream community and powerful people. This is because their paradigm-shifting thoughts and ideas on the subject, though more grounded in fact, threaten the goal of the global elite, which NSA whistleblower William Binney says, is “total population control.” The average person who gets a bachelor’s degree in science is trained to simply repeat the same old textbook rhetoric as to why evolution is the be all and end all of human existence, without actually looking into why the theory is highly questionable.

One of the latest dissenters is David Gelernter, a prominent scientist and distinguished professor of computer science at Yale University. He recently published an essay in the Claremont Review of Books explaining his objections to a premise behind Darwin’s theory.

He first points to the famous “Cambrian Explosion” which occurred half a billion years ago, in which a number of new organisms, including the first ever known animals, pop up suddenly in the fossil record over a period of approximately 70 million years. Apparently, this giant explosion of spontaneous life was followed by evolution, slow growth and “scanty fossils, mainly of single celled organisms, dating back to the origins of life roughly three and a half billions years ago.”

From here, he explains how Darwin’s theory predicts that new life forms evolve gradually from preceding ones. but if this is applied to the Cambrian creatures as well, it doesn’t work. The predecessors to the Cambrian creatures are missing, something that Darwin himself was disturbed by as well. Furthermore, even without this fact, many scientists have already used other aspects of the fossil record to demonstrate that Darwin’s theory is clearly wrong.

The Cambrian explosion had been unearthed, and beneath those Cambrian creatures their Precambrian predecessors should have been waiting – and weren’t. In fact, the fossil record as a whole lacked the upward-branching structure Darwin predicted….the ever-expanding fossil archives don’t look good for Darwin, who made clear and concrete predictions that have (so far) been falsified—according to many reputable paleontologists, anyway. When does the clock run out on those predictions? Never. But any thoughtful person must ask himself whether scientists today are looking for evidence that bears on Darwin, or looking to explain away evidence that contradicts him. There are some of each. Scientists are only human, and their thinking (like everyone else’s) is colored by emotion. (source)

The Genesis Of New Life Forms

His next point goes a little deeper. Many people point to the fact that variation occurs naturally among individuals and different traits are past on, this is something observable and something that we all know. Many scientists actually use this point as a proof for evolution, which doesn’t make much sense. According to proponents of the theory of evolution, natural variation is the consequence of random change or mutation to cells, to the genetic information within our cells that deal with reproduction. These cells pass on genetic change to the next generation, which, according to Darwinians, changes the future of the species and not just the individual.

The engine behind this thought, as Gelernter explains, is ‘change’ driven by the survival of the fittest and, obviously, lots and lots of time. He then goes on to ask a very crucial question: What exactly does generating new forms of life entail? Many within the field agree that generating a new shape of protein is the key to it. But does Darwinian evolution even purport to be able to do that? For Chris Williams, A Ph.D., Biochemistry Ohio State University, the full scope of Darwinian Evolution barely touches upon this important matter:

As a biochemist and software developer who works in genetic and metabolic screening, I am continually amazed by the incredible complexity of life. For example, each of us has a vast ‘computer program’ of six billion DNA bases in every cell that guided our development from a fertilized egg, specifies how to make more than 200 tissue types, and ties all this together in numerous highly functional organ systems. Few people outside of genetics or biochemistry realize that evolutionists still can provide no substantive details at all about the origin of life, and particularly the origin of genetic information in the first self-replicating organism. What genes did it require — or did it even have genes? How much DNA and RNA did it have — or did it even have nucleic acids? How did huge information-rich molecules arise before natural selection? Exactly how did the genetic code linking nucleic acids to amino acid sequence originate? Clearly the origin of life — the foundation of evolution – is still virtually all speculation, and little if no fact.

Intelligent Design

More and more, the evidence points to the great intelligence apparent in the system of life-creation. The reason that Darwinian Evolution is being left behind, and for many is obsolete, is because it is completely based on random, non-intelligent processes. Edward Peltzer Ph.D. Oceanography, University of California, San Diego (Scripps Institute), Associate Editor, Marine Chemistry, uses a clear real-life laboratory example to explain the need to posit the existence of an overriding ‘intelligence’ in order for things to make any sense:

As a chemist, the most fascinating issue for me revolves around the origin of life. Before life began, there was no biology, only chemistry — and chemistry is the same for all time. What works (or not) today, worked (or not) back in the beginning. So, our ideas about what happened on Earth prior to the emergence of life are eminently testable in the lab. And what we have seen thus far when the reactions are left unguided as they would be in the natural world is not much. Indeed, the decomposition reactions and competing reactions out distance the synthetic reactions by far. It is only when an intelligent agent (such as a scientist or graduate student) intervenes and “tweaks” the reactions conditions “just right” do we see any progress at all, and even then it is still quite limited and very far from where we need to get. Thus, it is the very chemistry that speaks of a need for something more than just time and chance. And whether that be simply a highly specified set of initial conditions (fine-tuning) or some form of continual guidance until life ultimately emerges is still unknown. But what we do know is the random chemical reactions are both woefully insufficient and are often working against the pathways needed to succeed. For these reasons I have serious doubts about whether the current Darwinian paradigm will ever make additional progress in this area.

Gelernter brings this conversation specifically to the generation of proteins:

Proteins are the special ops forces (or maybe the Marines) of living cells, except that they are common instead of rare; they do all the heavy lifting, all the tricky and critical assignments, in a dazzling range of roles. Proteins called enzymes catalyze all sorts of reactions and drive cellular metabolism. Other proteins (such as collagen) give cells shape and structure, like tent poles but in far more shapes. Nerve function, muscle function, and photosynthesis are all driven by proteins. And in doing these jobs and many others, the actual, 3-D shape of the protein molecule is important.

So, is the simple neo-Darwinian mechanism up to this task? Are random mutation plus natural selection sufficient to create new protein shapes?

Diving Into Proteins

Gelernter goes on to answer that question in great detail, and after going through the entire explanation he comes to what seems to be an inarguable conclusion. That the Theory of Evolution cannot, in any way, be a possible explanation for the generation of new proteins and mutations that are required for evolution to occur at all. This explanation is complex, but well worth it if you really want to understand how the ‘Theory of Evolution’ is refuted by the science of proteins:

How to make proteins is our first question. Proteins are chains: linear sequences of atom-groups, each bonded to the next. A protein molecule is based on a chain of amino acids; 150 elements is a “modest-sized” chain; the average is 250. Each link is chosen, ordinarily, from one of 20 amino acids. A chain of amino acids is a polypeptide—“peptide” being the type of chemical bond that joins one amino acid to the next. But this chain is only the starting point: chemical forces among the links make parts of the chain twist themselves into helices; others straighten out, and then, sometimes, jackknife repeatedly, like a carpenter’s rule, into flat sheets. Then the whole assemblage folds itself up like a complex sheet of origami paper. And the actual 3-D shape of the resulting molecule is (as I have said) important.

Imagine a 150-element protein as a chain of 150 beads, each bead chosen from 20 varieties. But: only certain chains will work. Only certain bead combinations will form themselves into stable, useful, well-shaped proteins.

So how hard is it to build a useful, well-shaped protein? Can you throw a bunch of amino acids together and assume that you will get something good? Or must you choose each element of the chain with painstaking care? It happens to be very hard to choose the right beads.

Inventing a new protein means inventing a new gene. (Enter, finally, genes, DNA etc., with suitable fanfare.) Genes spell out the links of a protein chain, amino acid by amino acid. Each gene is a segment of DNA, the world’s most admired macromolecule. DNA, of course, is the famous double helix or spiral staircase, where each step is a pair of nucleotides. As you read the nucleotides along one edge of the staircase (sitting on one step and bumping your way downwards to the next and the next), each group of three nucleotides along the way specifies an amino acid. Each three-nucleotide group is a codon, and the correspondence between codons and amino acids is the genetic code. (The four nucleotides in DNA are abbreviated T, A, C and G, and you can look up the code in a high school textbook: TTA and TTC stand for phenylalanine, TCT for serine, and so on.)

Your task is to invent a new gene by mutation—by the accidental change of one codon to a different codon. You have two possible starting points for this attempt. You could mutate an existing gene, or mutate gibberish. You have a choice because DNA actually consists of valid genes separated by long sequences of nonsense. Most biologists think that the nonsense sequences are the main source of new genes. If you tinker with a valid gene, you will almost certainly make it worse—to the point where its protein misfires and endangers (or kills) its organism—long before you start making it better. The gibberish sequences, on the other hand, sit on the sidelines without making proteins, and you can mutate them, so far as we know, without endangering anything. The mutated sequence can then be passed on to the next generation, where it can be mutated again. Thus mutations can accumulate on the sidelines without affecting the organism. But if you mutate your way to an actual, valid new gene, your new gene can create a new protein and thereby, potentially, play a role in evolution.

Mutations themselves enter the picture when DNA splits in half down the center of the staircase, thereby allowing the enclosing cell to split in half, and the encompassing organism to grow. Each half-staircase summons a matching set of nucleotides from the surrounding chemical soup; two complete new DNA molecules emerge. A mistake in this elegant replication process—the wrong nucleotide answering the call, a nucleotide typo—yields a mutation, either to a valid blueprint or a stretch of gibberish.

Building a Better Protein

Now at last we are ready to take Darwin out for a test drive. Starting with 150 links of gibberish, what are the chances that we can mutate our way to a useful new shape of protein? We can ask basically the same question in a more manageable way: what are the chances that a random 150-link sequence will create such a protein? Nonsense sequences are essentially random. Mutations are random. Make random changes to a random sequence and you get another random sequence. So, close your eyes, make 150 random choices from your 20 bead boxes and string up your beads in the order in which you chose them. What are the odds that you will come up with a useful new protein?

It’s easy to see that the total number of possible sequences is immense. It’s easy to believe (although non-chemists must take their colleagues’ word for it) that the subset of useful sequences—sequences that create real, usable proteins—is, in comparison, tiny. But we must know how immense and how tiny.

The total count of possible 150-link chains, where each link is chosen separately from 20 amino acids, is 20150. In other words, many. 20150 roughly equals 10195, and there are only 1080 atoms in the universe.

What proportion of these many polypeptides are useful proteins? Douglas Axe did a series of experiments to estimate how many 150-long chains are capable of stable folds—of reaching the final step in the protein-creation process (the folding) and of holding their shapes long enough to be useful. (Axe is a distinguished biologist with five-star breeding: he was a graduate student at Caltech, then joined the Centre for Protein Engineering at Cambridge. The biologists whose work Meyer discusses are mainly first-rate Establishment scientists.) He estimated that, of all 150-link amino acid sequences, 1 in 1074 will be capable of folding into a stable protein. To say that your chances are 1 in 1074 is no different, in practice, from saying that they are zero. It’s not surprising that your chances of hitting a stable protein that performs some useful function, and might therefore play a part in evolution, are even smaller. Axe puts them at 1 in 1077.

In other words: immense is so big, and tiny is so small, that neo-Darwinian evolution is—so far—a dead loss. Try to mutate your way from 150 links of gibberish to a working, useful protein and you are guaranteed to fail. Try it with ten mutations, a thousand, a million—you fail. The odds bury you. It can’t be done.

Proteins/Mutations Are One of Several Issues

Despite all of the scientific dogma that plagues this issue, proteins/mutations and lack of fossil evidence are simply the tip of the iceberg when it comes to finding faults found within the Theory of Evolution. There are many facts, information, science and new discoveries that would make one wonder how it’s even still being taught.

Furthermore, despite the fact that we get pounded with the idea that random mutation is ultimate truth within the mainstream, and that one is wrong for questioning it, there are a number of prominent scientists, who are actually getting together in large numbers to collectively refute Darwinism. A group of 500 scientists from several fields came together a few years to create “A Scientific Dissent From Darwinism,” as one examples. The issue is that these scientists are never getting any mainstream attention. But clearly there are some very intelligent people here.

The theory will be with us for a long time, exerting enormous cultural force. Darwin is no Newton. Newton’s physics survived Einstein and will always survive, because it explains the cases that dominate all of space-time except for the extreme ends of the spectrum, at the very smallest and largest scales. It’s just these most important cases, the ones we see all around us, that Darwin cannot explain. Yet his theory does explain cases of real significance. And Darwin’s intellectual daring will always be inspiring. The man will always be admired.

He now poses a final challenge. Whether biology will rise to this last one as well as it did to the first, when his theory upset every apple cart, remains to be seen. How cleanly and quickly can the field get over Darwin, and move on?—with due allowance for every Darwinist’s having to study all the evidence for himself? There is one of most important questions facing science in the 21st century.

Other Examples That Throw Off The Theory Of Evolution

Not long ago I wrote about a  recent paper published by 33 scientists in the Progress in Biophysics and Molecular Biology journal suggesting that the flourishing of life during the Cambrian era (Cambrian Explosion) originates from the stars is so fascinating.

“With the rapidly increasing number of exoplanets that have been discovered in the habitable zones of long-lived red dwarf stars (Gillon et al., 2016), the prospects for genetic exchanges between life-bearing Earth-like planets cannot be ignored. ” (The study)

There is a great little blurb from Cosmos Magazine, one of the few outlets who are talking about the study:

With 33 authors from a wide range of reputable universities and research institutes, the paper makes a seemingly incredible claim. A claim that if true, would have the most profound consequences for our understanding of the universe. Life, the paper argues, did not originate on the planet Earth.

The response?

Near silence.

The reasons for this are as fascinating as the evidence and claims advanced by the paper itself. Entitled “Cause of the Cambrian Explosion – Terrestrial or Cosmic?”, the publication revives a controversial idea concerning the origin of life, an idea stretching back to Ancient Greece, known as ‘panspermia.a’.

Academics like Francis Crick, an English scientist who co-discovered the structure of the DNA molecule (alongside James D. Watson), argues that there is no possible way that the DNA molecule could have originated on Earth. The generally accepted theory in this field, as explained above, is that we are the result of a bunch of molecules accidentally bumping into each other, creating life. However, according to Crick, we are the result of what is now known as Directed Panspermia. Crick and British chemist Leslie Orgel published their paper on it in July of 1973, hinting that we were brought here by chance, or by some sort of intelligence from somewhere else in the universe.

This is interesting, because then you can get into the lore of creation stories that exists within ancient cultures from around the world, one would be our relation to, for example, what many indigenous culture refer to as the ‘Star People.’

I’m not even going to go into all of the strange skeletal remains that have been completely left out of the record, like the remains of giants, for example.

The Takeaway

The agenda for the maintenance of the neo-Darwinian version of the ‘Theory of Evolution’ was nothing less than to move people away from the notion of an intelligent creator and towards a perception founded in scientific materialism. In this way, those who funded and controlled scientific activity on the planet would have tremendous power.

Darwin’s theory may have served humanity for a certain phase of our own evolution, but now it is holding us back. It’s time for all of us to pierce more deeply into an understanding of the nature of the creation of life if we are to become creators ourselves by studying the current evidence. As the group of 500 scientists asked, ‘How cleanly and quickly can the field get over Darwin, and move on?—with due allowance for every Darwinist’s having to study all the evidence for himself?’

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Cancer vaccine shown to be safe and effective has entered human trials

Cancer vaccine shown to be safe and effective has entered human trials 91
University of Montreal

A person, like any living creature, can be vaccinated against cancer, although this disease is fundamentally different from viral infections against which vaccines are traditionally used. 

This has been proven by scientists at Ohio University, who developed a methodology for the use of immune checkpoint inhibitors. Animal studies have shown 90% effectiveness of this therapy and complete safety for the body.

Cancer tumors are extremely insidious and have a defense mechanism against the body’s immune system in the form of the signaling protein PD-1. It is present on both healthy and cancerous cells, and is responsible for the friend-or-foe recognition procedure when immune B and T cells approach them. As long as PD-1 proteins in cancer cells and PD-L1 proteins in lymphocytes are working normally, the immune system simply ignores the infection, not seeing it as a target to attack.

The idea of ​​Dr. Pravin Kaumay, the developer of the inhibitors, is to interfere with the identification procedure. For this purpose, special monoclonal antibodies have been developed, which are injected into the body, seek out PD-1 proteins and settle on them, preventing proper contact with PD-L1 proteins. Lymphocytes cannot recognize these cells and automatically start the procedure for destroying them – the immune system itself begins to eradicate cancerous tumors in the body.

More importantly, blocking the signaling system destroys the usual comfortable environment for cancer cells, they are constantly threatened, cannot grow and spread throughout the body. 

This is the beneficial effect of vaccination with inhibitors – this therapy is called PD1-Vaxx. The technology has been thoroughly tested, it uses second-generation inhibitors, which are much more effective. The first human patients have already been recruited in the US and Australia to test PD1-Vaxx.

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A lack of oxygen leads to aging, excess oxygen leads to immortality

A lack of oxygen leads to aging, excess oxygen leads to immortality 92

Israeli scientists claim that by using only oxygen, they have successfully reversed the biological aging process.

A recent study conducted by Tel Aviv University professor Shai Efrati and a team at Shamir Medical Center showed that when healthy adults over 64 were placed in a pressurized chamber and given pure oxygen for 90 minutes a day, five days a week and within three months, the aging process not only slowed down, but actually changed in the opposite direction.

In particular, a study published in the peer-reviewed journal Aging focused on whether this oxygen enrichment process could reverse two key indicators of biological aging: the contraction of DNA telomeres and the accumulation of senescent cells in tissues. Telomeres are located at the ends of a chromosome, consist of repeating sequences of non-coding DNA, and serve as caps to protect the chromosome from damage during replication.

A lack of oxygen leads to aging, excess oxygen leads to immortality 93

Each time replication occurs, these bumpers take a hit, making the chromosomes shorter and shorter. Once the telomere reaches a certain length, the cell can no longer replicate, which leads to aging: malfunctioning of cells, which ultimately leads to cognitive or other age-related impairments and even diseases such as cancer.

About 35 adults over the age of 64 took part in the study and applied hyperbaric oxygen therapy, using 100% oxygen at an ambient pressure greater than one absolute atmosphere to increase the amount of oxygen dissolved in body tissues.

A lack of oxygen leads to aging, excess oxygen leads to immortality 94

Every 20 minutes, participants were asked to remove their masks for five minutes, returning oxygen to normal levels.

However, during this period, the researchers saw that fluctuations in free oxygen concentration were interpreted at the cellular level as a lack of oxygen – rather than interpreting the absolute level of oxygen.

In other words, repeated intermittent hyperoxic (increased oxygen levels) exposure induced many of the mediators and cellular mechanisms that are usually induced during hypoxia (low oxygen levels). Dr. Efrati calls this the hyperoxic-hypoxic paradox.

“The fluctuations in the oxygen level that we have created is what matters,” he told The Jerusalem Post. “During this process, there is, as it were, a lack of oxygen, which causes cell regeneration.”

The practical consequences of therapy include increased attention, processing speed, and executive function, which tend to decrease with age and about which more than 50% of people over the age of 60 express concern.

According to the study, the changes were equivalent to having the participants’ bodies at the cellular level go back 25 years.

“We’re not [just] slowing aging – we’re moving back in time,” says Dr. Efrati. Efrati has been studying aging for ten years and runs Aviva clinics in Florida. The study, he said, is evidence that the cellular basis of the aging process can be reversed, adding that it “offers hope and opens up the opportunity for many young scientists to target aging as a reversible disease.”

It could also allow doctors and scientists to find a way to control telomere length and develop drugs to help them grow again when needed. But will it make people live longer?

The duration of the effect has yet to be determined in the long term, says Efrati. But “probably yes.” We know people with shorter telomeres die earlier, so that makes sense.”

At the moment, the only drawback of the study is the limited sample size, that is, so far relatively few people have participated in the experiment, but over time, scientists will solve this problem too.

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Cyberfarm 2077. YouTuber showed what a Russian cyber village could look like

Cyberfarm 2077. YouTuber showed what a Russian cyber village could look like 95
Photo: video screenshot youtube.com/birchpunk

On November 19, the day the Cyberpunk 2077 video game was supposed to be released, a five-minute video about a Russian cyber village appeared on YouTube. It combines the aesthetics of cyberpunk with the life of the Russian provinces.

Cyberfarm 2077. YouTuber showed what a Russian cyber village could look like 96

Russian director and blogger Sergei Vasiliev made a short film that is an excerpt from the life of a fictional Russian village with the technologies of the future. The short film was shot in the style of a video blog. 

“They say that Russia is a technically backward country, there are no roads, no robotics, rockets do not fly, and mail takes too long. [This is not true],” the creators say.

The main character, a farmer called Nikolai, talks about his cyber farm, where three androids work, and invites new workers to his business. The robots milk the cows, walk them and go to fetch water. Instead of a cesspool, there is a black hole in the toilets, and mail is delivered by flying wagons of the Russian Post. New technologies have also solved the problem of Russian roads – cars now fly over it, but even there are holes in the air.

The authors of the short film said that a “creative association of enthusiasts” worked on the creative, and now they “plan to release further videos in the same universe.”

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